Bone Marrow Custard

Bone Marrow Custard

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My version is based off this recipe from Kasey Culinary Adventures . Learn about the benefits of bone marrow in one of our articles! Ingredients

5 lbs beef marrow bones, unsliced

1 cup of cream

3 egg yolks

3 whole eggs

1 Tbs vanilla extract

2 teaspoons honey or maple syrup — optional

½ teaspoon of sea salt

1 tablespoon of cinnamon, or to taste


In bowl, combine marrow (try not to include the oil), 1 cup cream, 3 egg yolks, 3 whole eggs, 2 tsp honey or maple syrup (optional), cinnamon to taste, dash of sea salt. Blend all together.

Pour into custard cups or cake pans (I used 2, 5” cake pans) and place those in a water bath.

In oven preheated to 350°F, bake until center is set. My pans took 40 minutes. Turn off oven and open door, allowing water bath to cool.

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This is a Web version of 28-page printed informational 28-page printed informational booklet . You may order printed copies via our Store.

The Weston A. Price Foundation only accepts contributions from members and/or private donations, and does not accept funds from the meat or dairy industries. Life in all its splendor is Mother Nature obeyed. – Weston A. Price, DDS


About Dr. Weston A. Price
Characteristics of Traditional Diets – 11 Principles
Dietary Guidelines
Dietary Dangers
Confused about Fats?
The Many Roles of Saturated Fats
The Fat-Soluble Activators
What’s Wrong With “Politically Correct” Nutrition?
Traditional vs. Modern Diets
Myths and Truths About Nutrition
Myths and Truths About Soy
Soy Infant Formula: Birth Control Pills for Babies Coronary Heart Disease: What the Expert Say Principles of Holistic Dentistry The Weston A. Price Foundation Become a Member of the Weston A. Price Foundation About Dr. Weston A. Price In the early 1930s, a Cleveland dentist named Weston A. Price (1870-1948) began a series of unique investigations. His portrait on the left is provided courtesy of the Price-Pottenger Nutrition Foundation who owns the copyright.For over ten years, he traveled to isolated parts of the globe to study the health of populations untouched by western civilization. His goal was to discover the factors responsible for good dental health. His studies revealed that dental caries and deformed dental arches resulting in crowded, crooked teeth are the result of nutritional deficiencies, not inherited genetic defects. The groups Price studied included remote villages in Switzerland, Gaelic communities in the Outer Hebrides, indigenous peoples of North and South America, Melanesian and Polynesian South Sea Islanders, African tribes, Australian Aborigines and New Zealand Maori. Wherever he went, Dr. Price found that beautiful straight teeth, freedom from decay, good physiques, resistance to disease and fine characters were typical of native groups on their traditional diets, rich in essential nutrients.When Dr. Price analyzed the foods used by isolated peoples he found that, in comparison to the American diet of his day, they provided at least four times the water-soluble vitamins, calcium and other minerals, and at least TEN times the fat-soluble vitamins, from animal foods such as butter, fish eggs, shellfish, organ meats, eggs and animal fats—the very cholesterol-rich foods now shunned by the American public as unhealthful. These healthy traditional peoples knew instinctively what scientists of Dr. Price’s day had recently discovered—that these fat-soluble vitamins, vitamins A and D, were vital to health because they acted as catalysts to mineral absorption and protein utilization. Without them, we cannot absorb minerals, no matter how abundant they may be in our food. Dr. Price discovered an additional fat-soluble nutrient, which he labeled Activator X, that is present in fish livers and shellfish, and organ meats and butter from cows eating rapidly growing green grass in the spring and fall. All indigenous groups had a source of Activator X, now thought to be vitamin K2, in their diets.The isolated groups Dr. Price investigated understood the importance of preconceptual nutrition for both parents. Many tribes required a period of special feeding before conception, in which nutrient-dense animal foods were given to young men and women. These same foods were considered important for pregnant and lactating women and growing children. Price discovered them to be particularly rich in minerals and in the fat-soluble activators found only in animal fats.The isolated people Price photographed—with their fine bodies, ease of reproduction, emotional stability and freedom from degenerative ills—stand in sharp contrast to civilized moderns subsisting on the “displacing foods of modern commerce,” including sugar, white flour, pasteurized milk, lowfat foods, vegetable oils and convenience items filled with extenders and additives.The discoveries and conclusions of Dr. Price are presented in his classic volume, Nutrition and Physical Degeneration. The book contains striking photographs of handsome, healthy, indigenous people that illustrate in an unforgettable way the physical degeneration that occurs when human groups abandon nourishing traditional diets in favor of modern convenience foods.The photographs Dr. Weston Price took illustrate the difference in facial structure between those on native diets and those whose parents had adopted the “civilized” diets of devitalized processed foods. The “primitive” Seminoles pictured on the left have wide, attractive faces with plenty of room for the dental arches. The “modernized” Seminole girl on the right, born to parents who had abandoned their traditional diets, has a narrowed face, crowded teeth and a reduced immunity to disease. Photos courtesy of the Price-Pottenger Nutrition Foundation , who owns the copyrights. All rights reserved. Characteristics of Traditional Diets – 11 Principles> The diets of healthy, nonindustrialized peoples contain no refined or denatured foods or ingredients, such as refined sugar or high fructose corn syrup; white flour; canned foods; pasteurized, homogenized, skim or lowfat milk; refined or hydrogenated vegetable oils; protein powders; synthetic vitamins; or toxic additives and artificial colorings. All traditional cultures consume some sort of animal food, such as fish and shellfish; land and water fowl; land and sea mammals; […]


Mexico City sinking at “unstoppable rate” of up to 20 inches a year, study finds

Mexico City sinking at “unstoppable rate” of up to 20 inches a year, study finds

( Natural News ) A recent study found that Mexico City is sinking at a rate of up to 20 inches per year . Researchers explained that this unstoppable plunge occurs because the ground on which the city rests is rapidly compacting after being drained of water by decades of groundwater extraction.

The Metropolitan Area of the Valley of Mexico is built on what was once Lake Texcoco, home of the Aztec capital Tenochtitlan. Also called the Greater Mexico City, the crowded metropolis encompasses Mexico City and other surrounding areas. It obtains more than three-quarters of its drinking water from beneath the ground. But water extraction in the region dates back several centuries, beginning during colonial Mexico .

This long-term pumping pushed the groundwater further underground, drying the lake bed so much that clay sheets began to crack and compress. With the earth repacking itself more tightly, the ground started to shrink and subside.

Authorities first noticed that Greater Mexico City was sinking in 1900 when the metro was sliding at a rate of around 3.5 inches a year. When the government placed a cap on water extraction in the late 1950s, it was sinking 11 inches a year.

The cap slowed the rate of sinking back to 3.5 inches a year but failed to eradicate the problem. In the years since, land subsidence worsened as the metro’s population ballooned to 21 million and urbanization increased exponentially.

The study, which was published March in the Journal of Geophysical Research: Solid Earth , showed that parts of the city, including the historic downtown in Mexico City, are now sinking up to 16 inches a year. In the underdeveloped northeast, the ground is dipping 20 inches a year. If the northeast were to become more industrialized, land subsidence would get even worse. (Related: The San Joaquin Valley is rapidly sinking due to groundwater extraction… some areas have fallen 28 feet… and it’s accelerating .)

The aquifer system beneath the metro was 17 percent compacted as of 2020. After analyzing more than a century of data, including recent satellite information, researchers predicted that the aquifer system would compact further to 30 percent, causing the metro to sink almost 100 feet in the next century.

The lost elevation is irreversible, leaving the region vulnerable to plenty of disasters. For one, subsidence makes the metro more prone to heavy flooding, which, in turn, can cause water shortages since floods can pollute the groundwater. Subsidence in the metro also fractures the ground , which previously damaged buildings, historical sites, sewers, and gas and water lines. This fracturing can also open up the earth to contaminated surface water, which can further restrict access to potable water.

“The stage is set for a dual water and subsidence crisis if no drastic water management actions are implemented,” the researchers concluded in their paper. What is land subsidence?

According to the U.S. Geological Survey , land subsidence occurs when large amounts of groundwater are withdrawn from certain types of rocks, such as fine-grained sediments. Rocks compact because groundwater is partly responsible for holding the ground up. When sufficient amounts of groundwater is withdrawn, rocks fall in on itself, causing the ground to sink.

Land subsidence affects more than 17,000 square miles in the U.S., or roughly the size of New Hampshire and Vermont combined. More than 80 percent of these sank due to the exploitation of groundwater, as well as the increased development of land and water resources.

California experiences the greatest losses in elevation among 45 states affected by land subsidence. In the San Joaquin Valley, some areas sank by as much as 28 feet since record-keeping began. Water pumping is intensive in the region, which is one of the most productive agricultural lands in the country. Over time, over-pumping dramatically reduced groundwater levels, drying out the aquifer system and causing it to compact.

Visit to learn more about how human activities like intensive agriculture damage the environment.

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( Natural News ) Researchers at the University of Washington found that cadmium exposure, combined with a genetic predisposition to Alzheimer’s disease, might accelerate cognitive decline. The researchers arrived at this finding after exposing mice with an Alzheimer’s risk gene to cadmium. They detailed their findings in a paper published in the journal Toxicological Sciences . The effects of cadmium plus an Alzheimer’s risk gene

The so-called human apolipoprotein E (APOE) gene codes for a protein called apolipoprotein. This combines with fats to form lipoproteins, which carry cholesterol through the bloodstream. The APOE4 gene is a variant of the APOE gene. It significantly raises a person’s risk of developing Alzheimer’s , which is why it is considered an Alzheimer’s risk gene.

For their study, the researchers wanted to know how being carrier of this gene, combined with exposure to cadmium, affects cognition. Cadmium is a heavy metal that is extremely toxic to the brain. It occurs naturally and is extracted during the production of copper, lead and zinc. Cigarette smoke and polluted air also contain this toxin.

Over a period of 14 weeks, the researchers gave a group of mice with the APOE4 or the APOE3 gene water that contained low doses of cadmium. APOE3 is another variant of the APOE gene. It is widespread, with around half of the population carrying it.

The maximum amount of cadmium that the mice ingested was equivalent to the amount that Americans have in their blood, including people who never smoked .

As part of their experiment, the researchers put the rats through standard novel object location tests and T-maze tests, both of which engage the hippocampus, a region of the brain that is crucial for learning and memory. The hippocampus is one of the brain areas that is damaged the most during the early stages of Alzheimer’s.

The mice that ingested cadmium performed worse in the novel object location tests than untreated mice, indicating poorer short-term spatial working memory. This deficit in memory appeared earlier in mice with the APOE4 gene than those with APOE3, and earlier […]


Animal study reveals cadmium exposure and Alzheimer’s risk gene may be linked to cognitive impairment

Animal study reveals cadmium exposure and Alzheimer’s risk gene may be linked to cognitive impairment

( Natural News ) Researchers at the University of Washington found that cadmium exposure, combined with a genetic predisposition to Alzheimer’s disease, might accelerate cognitive decline. The researchers arrived at this finding after exposing mice with an Alzheimer’s risk gene to cadmium. They detailed their findings in a paper published in the journal Toxicological Sciences . The effects of cadmium plus an Alzheimer’s risk gene

The so-called human apolipoprotein E (APOE) gene codes for a protein called apolipoprotein. This combines with fats to form lipoproteins, which carry cholesterol through the bloodstream. The APOE4 gene is a variant of the APOE gene. It significantly raises a person’s risk of developing Alzheimer’s , which is why it is considered an Alzheimer’s risk gene.

For their study, the researchers wanted to know how being carrier of this gene, combined with exposure to cadmium, affects cognition. Cadmium is a heavy metal that is extremely toxic to the brain. It occurs naturally and is extracted during the production of copper, lead and zinc. Cigarette smoke and polluted air also contain this toxin.

Over a period of 14 weeks, the researchers gave a group of mice with the APOE4 or the APOE3 gene water that contained low doses of cadmium. APOE3 is another variant of the APOE gene. It is widespread, with around half of the population carrying it.

The maximum amount of cadmium that the mice ingested was equivalent to the amount that Americans have in their blood, including people who never smoked .

As part of their experiment, the researchers put the rats through standard novel object location tests and T-maze tests, both of which engage the hippocampus, a region of the brain that is crucial for learning and memory. The hippocampus is one of the brain areas that is damaged the most during the early stages of Alzheimer’s.

The mice that ingested cadmium performed worse in the novel object location tests than untreated mice, indicating poorer short-term spatial working memory. This deficit in memory appeared earlier in mice with the APOE4 gene than those with APOE3, and earlier in males than females with the same genetic makeup.

Later in life, mice with the APOE4 gene performed worse in the T-maze test than those with APOE3. This indicated a reduced tendency to explore new environments and therefore poorer short-term spatial working memory. In addition, the researchers found that cadmium exposure negatively impacts nerve cell development in the hippocampus of male mice with the APOE4 gene. Cadmium pools in the brain due to Alzheimer’s risk gene

These findings led the researchers to conclude that interactions between the APOE4 gene and cadmium accelerates cognitive decline, with young male mice being more susceptible to this effect than young females. The researchers pointed to impaired nerve cell development in the hippocampus as one of the underlying mechanisms, which possibly occurs because the APOE4 gene lets cadmium into the brain.

“It is possible that APOE4 may cause leakage on the blood-brain barrier and lead to a higher degree of cadmium accumulation in the APOE4 brain,” the researchers wrote. (Related: Cadmium could have adverse effects on brain development, reducing neurogenesis .)

Zhengui Xia, a professor of toxicology at Washington University and one of the study’s researchers, commented: “Exposure to cadmium through our daily lives could have a detrimental effect on our cognition. If you have the APOE4 gene, the risk is significantly higher.”

“This heavy metal is bad for you,” she added.

Follow to learn more about the health risks of cadmium and other heavy metals.

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Matcha Vs Green Tea: What Is The Difference?

One of the most powerful drinks and a healthy beverage green tea is something which has been popularised for years now and is known for the various health benefits it comes with from keeping your heart healthy to aiding weight loss and from improving the brain function to helping prevent cancer this drink can do it all. While we all were busy enjoying the benefits of green tea and sipping a hot cup in the morning a new similar kind of beverage, Matcha emerged and has taken over the internet and especially the social media websites. Here let us understand about the health benefits of both these drinks, the difference between them and some quick and amazing recipes that you can enjoy, as explained below by Nutritionist And Public Health Expert Swati Bathwal . Green Tea

here could be nothing better and healthier than starting your day with a nice cup of green tea along with some honey and a few drops of lemon. Green tea is known to be one of the most healthy beverages which helps to detoxify your body, is good for your skin and hair and is also proven to lower the risk of various types of cancers. Let us take a closer look at the health benefits of drinking green tea.

Also Read: 5 Health Benefits Of Drinking Green Tea Everyday 1# Prevents Type 2 Diabetes

As type 2 diabetes has been increasing rapidly at a very high rate all over the world, an easy solution which helps to improve the insulin sensitivity green tea helps to reduce the blood sugar levels in the body which is usually increased by the body’s inability to produce insulin naturally which is the reason behind the body’s elevating blood sugar levels. 2# Aids weight loss

Rich in antioxidants and low in calories , green tea is a magical beverage that aids fat burning and weight loss due to the presence of catechins compounds in it by boosting the metabolic rate and improving the overall digestive system. Green tea helps to reduce the dangerous fat in the abdominal region by boosting the metabolism rate in a short span of time and helps in reducing the weight size, body fat, belly fat, body weight and the overall body fat percentage as well. 3# Lowers the risk of cardiovascular diseases

With heart attacks being one of the leading causes of deaths world wide, green tea is a beverage that can lower the risk of heart attacks and other cardiovascular diseases is consumed on a regular basis. This light and aromatic drink helps in lowering the lower LDL cholesterol levels and triglycerides which as a result lowers the risk of getting a heart attack or stroke hence is considered to be healthy for heart . 4# Helps prevent cancer

Not just by preventing but also by lowering the risk of a chronic disease like cancer green tea helps in reducing inflammation and prevent various types of cancer like breast cancer, prostate cancer, mouth cancer, stomach cancer, kidney cancer, colorectal cancer, ovarian cancer , cancer in mammary glands and pancreas, Cancer is a deadly disease caused by the uncontrolled growth of cancer cells in the body is becoming one of the leading cause of death worldwide. Consuming green tea on a regular basis can actually reduce the risk of getting this chronic deadly disease as it consists of antioxidants and is rich in polyphenols, it helps in reducing inflammation and fight the inflammation causing oxidative stress. Green Tea Recipe

As we now know about the different health benefits a cup of green tea comes with, let us take a quick run through the quick and green tea recipe that you can enjoy early in the morning.

Ingredients Green tea bag

1 tablespoon of honey

3-4 drops of lemon juice

1 cup water

How to Make? Take a cup of water and boil it on medium flame.

Once boiled, transfer the water to a cup and add honey and lemon juice to it.

Stir it well and add the tea bag to the cup and let it steep in for 3-5 minutes.


A Japanese beverage that has skyrocketed popularly lately, Matcha comes in many forms and is an ingredient which has various uses and health benefits. This beverage can be found on most of the coffee shops around and hasn’t failed to show its presence all over the social media websites. With high antioxidant content matcha can help to protect your lover and might also be helpful in preventing cancer. Let us dig a little deeper into the health benefits this trending drink has. 1# Helps to protect the Liver

Matcha decreases the risk of liver diseases by preventing liver damage. Researches have shown that matcha supports the liver’s natural detoxification process and is proven to control the liver enzymes as high amounts of liver enzymes can be dangerous for the liver and can cause damage. 2# Boosts Brain Functioning

Rich in polyphenols matcha is also known as the brain booster which when consumed on a regular basis helps to enhance memory, improve attention span and faster the reaction time. Its nutritional properties act as a nootropic that helps in boosting the cognitive functions of the body like attention, memory, concentration and motivation along with making the brain work as a vasodilator and makes it healthier by increasing the flow of oxygen to the brain. 3# Promotes Heart Health

Known for promoting a healthy heart by reducing the risk of heart attacks and strokes matcha helps in reducing both cholesterol LDL levels and triglycerides. The nutrients present in matcha can help in achieving peace of mind, elevating ood, weight loss in people suffering from obesity and the presence of antioxidants, caffeine, amino acids, vitamins, minerals and fibre can help to fight morbid illness. Matcha Latte Recipe

An easy recipe to prepare a rejuvenating and nutritious cup of matcha latte to enjoy […]


Study: Extreme adiposity associated with obesity starts with inflammation in the brain

Study: Extreme adiposity associated with obesity starts with inflammation in the brain

( Natural News ) Obesity is a metabolic disease that is now considered to be an epidemic. In the United States, obesity is a major contributor to some of the leading causes of death among Americans, which include heart disease, diabetes and some types of cancer.

The main defining feature of obesity is the excessive accumulation and storage of body fat . Researchers have identified several contributing factors to this abnormal event, such as genetics and behavioral, metabolic and hormonal influences on body weight. But the culprit most commonly associated with obesity is the intake of too many calories coupled with a lack of exercise or physical activity. As pointed out by many studies, calories that are not burned through physical activities are stored by the body as fat.

Not surprisingly, the adult obesity rate in the U.S. is considerably high , exceeding 40 percent, because the standard American diet is made up of mostly sugar-laden beverages and fatty, highly processed foods. Aside from providing very little nutrients, research has found that these high-calorie foods make a person eat more than he actually needs because they are not satiating. This, combined with a sedentary lifestyle, is behind the alarming rise in both childhood and adult obesity rates seen in the country. The link between inflammation and obesity

In an effort to understand what drives obesity at the molecular level, researchers at Ewha Womans University in South Korea decided to explore the mechanisms that lead to obesity . Their study, which appeared in the journal Nutrition Research, focused mainly on inflammation. Chronic inflammation has been linked to obesity and other serious conditions like heart disease and cancer.

According to an article published in Science , alterations in the gene sequence and increased expression of RIPK1 , a key regulator of inflammation, are some of the main contributors to metabolic disease. When researchers inhibited the expression of RIPK1 in mice on a high-fat diet, they found that it not only reduced inflammatory responses, but also body weight and fat accumulation. This suggests that this inflammatory gene plays a crucial role in the development of obesity .

For their study, however, the Korean researchers focused on a mechanism that has only been proposed recently. This mechanism also involves inflammation, albeit in the brain instead of adipose tissue. Recent studies suggest that inflammation in the hypothalamus, the part of the brain that governs systemic metabolism, may also be a driving force behind obesity. (Related: Researchers conclude that drinking soda during pregnancy causes obesity in offspring .)

The researchers hypothesized that a high-fat diet could trigger metabolic inflammation via transcriptional changes (i.e., changes in gene expression) in the hypothalamus. To test their hypothesis, they characterized obesity-related in vivo transcriptional alterations in the hypothalamus and their effects on functional networks.

The researchers fed two groups of mice either a control diet or a high-fat diet for 20 weeks before conducting microarray and gene ontology analyses of the animals’ hypothalami. They reported that in the brains of mice on a high-fat diet, immune-related pathways such as inflammatory signaling were overly activated. This was not the case with mice on the control diet.

Meanwhile, in mice deficient in leptin — the hormone released by fat cells to tell the brain, particularly the hypothalamus, to suppress appetite — the researchers found that genes involved in inflammatory pathways and cancer pathways were highly expressed. They noted a similar overexpression in the hypothalami of mice on a high-fat diet, which confirms their hypothesis that brain inflammation is heavily involved in obesity.

Based on these findings, the researchers concluded that, rather than dietary fat and genetic mutation, inflammation in the hypothalamus is likely to be the cause of excessive fat accumulation associated with obesity.

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The science of why the sun makes us feel so happy

The science of why the sun makes us feel so happy

With the hottest day of the year so far coinciding with the last step in the Government’s lockdown roadmap, it is clear that the sun has put many of us in a cheerful mood, especially after the dark winter spent indoors.

Although we are not able to go to many of the popular sun spots across the globe, this summer has shown that we are able to soak up the rays in the UK and that provides us with the feel good factor that we may have missed in the past 18 months.

But what is it about the sunshine that makes us feel so happy? Here is the science behind why those rays are so good for us. It boosts our mood

Most of us will agree that it’s hard to feel unhappy in the sun. This is down to the link between sunlight and our serotonin levels – the hormone that makes us feel happy. It’s also why people are more likely to develop Seasonal Affective Disorder (SAD) when the shorter autumn days arrive and we creep into winter with fewer daylight hours.

There is plenty of research to back up this idea. One study undertaken in Australia found that people had higher levels of serotonin on bright sunny days than cloudy ones. Increased levels of this hormone generally lead to greater feelings of satisfaction and calmness and lower levels of depression and anxiety.

There is even research to show that people who use tanning beds may experience more frequent feelings of euphoria, which could explain why people develop a dependence on regular sessions. Although the connection isn’t entirely established yet, researchers speculate this could be down to the way UV light forces melanocytes, the cells that produce dark pigment in skin, to release endorphins. However, most experts also agree that the increased sun cancer risk negates the feel good factor. Sun improves our sleep

Regular exposure to sunlight encourages the production of melatonin – the hormone which helps to regulate the body’s sleep-wake cycle. This encourages feelings of drowsiness, allowing us to drift off easier at night, which leads to us feeling happier in the day. Melatonin also helps to regulate our circadian rhythm – the body’s internal clock that signals when to be alert and when to rest – which can be thrown out of sync by exposure to blue light from technology, disrupted work patterns and light pollution.

In turn, this allows us to feel happier. Research shows that our amygdala – the emotional part of the brain – is significantly more reactive after a bad night’s sleep, meaning we are more likely to feel cranky throughout the day if we have spent the night tossing and turning. Time spent in the sun can help us sleep soundly. Our sex drive is given a lift

Believe it or not, even our sex drive is affected by time spent in the sun, so a spring heatwave is good news for those who have found their libido dampened somewhat in lockdown. Researchers at Medical University of Graz in Austria found that spending just one hour in the sun can boost a man’s testosterone levels by 69 per cent. In turn, this helps to balance mood, sex drive and cognitive function. The experts put this down to the role of vitamin D, which is produced after exposure to sunlight .

It’s the same situation for women. Researchers in China, who conducted a study on post-menopausal women, identified a link between low levels of vitamin D and low levels of oestrogen, the female sex hormone. Your bones will be given a boost

Vitamin D is also crucial for helping our body to absorb calcium, which is responsible for strengthening your bones. A lack of vitamin D has been associated with both osteoporosis, rickets and autoimmune diseases, such as rheumatoid arthritis (RA).

A review by the Cochrane Library found that the rates of falls in elderly people – which are partly down to the effects of brittle bones – could be cut by more than a quarter if the elderly were given supplements of vitamin D. However, in recent years many studies have questioned how effective supplements are in reducing rates of osteoporosis.

That’s not to say that sunlight can’t help though: more than 90 per cent of a person’s vitamin D requirement tends to come from casual exposure to sunlight, making it the best source of the nutrient. So how much exposure do we need to boost our health? On average, experts believe we should be aiming for 10–30 minutes of midday sunlight, several times per week. So make sure you head outside for a walk today to get a boost of bone-strengthening vitamin D. It improves midlife brain health

While most of the research around sunlight and the brain has focused on serotonin levels, a dose of vitamin D could also be good for our intellect. In 2009, scientists from the University of Manchester found that higher levels of vitamin D are linked with improved mental ability in middle-aged and older men. Men in the study were tested for memory and speed recollection, as well as for mood and physical activity levels, before their blood samples were taken. The researchers found that men with higher levels of vitamin D performed consistently better than those with lower levels. Your eyes need sunlight

Dr Rangan Chatterjee, GP and author of Feel Great, Lose Weight , explains that light is measured in a unit called lux: if we spend 20 minutes outside – even on a cloudy overcast day – we are exposed to around 10,000 lux, compared with 500 lux if we spend time indoors. This is particularly important for children. Researchers at King’s College London, the London School of Hygiene and Tropical Medicine, found that regular exposure to sunlight lowered the risk of nearsightedness – or myopia – in children and young adults by helping the eye produce dopamine, which aids in healthy eye development.

Exposure to natural light can also help […]


Breakthrough brain implant allows paralysed man to ‘speak’ by transcribing his thoughts

Breakthrough brain implant allows paralysed man to ‘speak’ by transcribing his thoughts

Paralyzed Speech Device (Photographs©2017 Barbara Ries. All rights reserved. 415-460-1440) An experimental brain implant that translates brain signals into words has been a success, say researchers in California.

It’s a major step towards the development of a technology that could help people communicate by thinking, potentially changing the lives of those who lose the ability to speak through injury or illness.

Scientists at the University of California, San Francisco, worked with a man in his 30s who suffered a paralysing stroke more than 15 years ago and lost the ability to speak, reports the Wall Street Journal . He agreed to have electrodes surgically attached to the outer surface of his brain to test the neuroprosthesis.

The experiment was detailed in a paper published in the New England Journal of Medicine. Over the course of 50 separate sessions, the researchers recorded the man’s brain activity as he observed words displayed on a screen and imagined saying them aloud.

The researchers said they could identify the word the man was saying almost half the time, which rose to 76% when the scientists incorporated word-prediction algorithms.

“To our knowledge, this is the first successful demonstration of direct decoding of full words from the brain activity of someone who is paralyzed and cannot speak,” said neurosurgeon Dr. Eddie Chang, the paper’s senior author. “It shows strong promise to restore communication by tapping into the brain’s natural speech machinery.”

Experts say the high error rate, limited vocabulary – this study used just 50 words – and the time it takes to train the system to recognise imagined words mean there’s still a long way to go before the technology could be used practically in the real world.

However, the experiment has shown that the brain region responsible for speech continues to function even years after the ability to speak has been lost and that computers can be taught to decode full words from brain activity.

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Here’s what you need to know about the must-have supplement for patients with Hashimoto’s low thyroid

Here’s what you need to know about the must-have supplement for patients with Hashimoto’s low thyroid

Stock image | Photo by spukkato/iStock/Getty Images Plus, St. George News LATEST STORIES

CONTRIBUTED CONTENT — Antioxidants are a buzzword in health circles that sometimes deliver empty promises. But when it comes to autoimmune Hashimoto’s hypothyroidism, one antioxidant is a must-have in your protocol kit: Glutathione. Stock image | Photo by Inside Creative House/iStock/Getty Images Plus, St. George News Glutathione is considered the body’s master antioxidant, and at RedRiver Health and Wellness Center , it’s the supplement we tell our patients they need it to dampen autoimmune response and lower the risk of developing new autoimmune diseases.

Glutathione protects cells from damage, supports general detoxification, acts as a natural chelator for toxic heavy metals and environmental toxins and supports healthy immune system function. Glutathione works by protecting energy-producing factories inside cells called mitochondria.

Antioxidants are molecules that inhibit other molecules from going through oxidation, a chemical reaction that produces toxins called free radicals. Free radicals are unstable molecules that occur naturally but also enter our bodies through toxins in food, air, water and even medications. Left unchecked, free radicals damage cells, destabilize the immune system and contribute to the development of serious health problems.

Glutathione is a compound made by the body that protects cells and tissues from damage by free radicals. While the body makes glutathione, it can also be supplemented in absorbable forms or via nutrients that boost glutathione production.

Normally, our bodies should make enough glutathione to protect us. However, it’s common for glutathione to drop too low in our modern world. Even if we lead a very clean, nontoxic life, we cope with thousands of toxic chemicals in our daily environment, our food and our water. Sugary diets full of processed foods, food intolerances, leaky gut and undiagnosed infections are other examples of things that can deplete glutathione due to chronic inflammatory assaults on the cells.

Glutathione levels also decrease as we age, and our need for supplemental glutathione increases significantly. When glutathione production drops, you’re more vulnerable to developing autoimmune disease , chronic pain, chemical sensitivities, leaky gut and other immune-related disorders.

In fact, glutathione depletion is linked with a number of disease states and groups including the following: Aging.

Athletic overtraining.

Major injuries and trauma.

Patients with wasting diseases such as HIV /AIDS.

Lung cancer.

Gut-based diseases such as Crohn’s and ulcerative colitis.

Chronic fatigue syndrome.

Alcoholism and fatty liver disease.

Diabetes and low glucose tolerance.


One of the most important things you can do to improve glutathione status is to remove or mitigate stressors that deplete glutathione. These may include lack of sleep, smoking, food intolerances, diets high in sugars and processed foods, excess alcohol intake and hormone or immune imbalances.

If you have Hashimoto’s , this typically also means going on a gluten-free diet, as many studies show a connection between Hashimoto’s hypothyroidism and a gluten intolerance or celiac disease.

Additionally, research shows a link between poor glutathione status and autoimmune Hashimoto’s hypothyroidism .

Recycle glutathione to manage Hashimoto’s hypothyroidism

One of the most effective approaches to dampen autoimmune-related inflammation is to support your body’s ability to recycle glutathione. Recycling glutathione means your body takes existing glutathione that has already been used in self-defense and rebuilds it so it can work again to protect the body.

For glutathione to be recycled, it must be reduced. There are two main forms of glutathione in the body: reduced glutathione and oxidized glutathione.

When there is sufficient reduced glutathione in the cells, they sacrifice themselves to free radicals to protect cellular mitochondria. An enzyme called glutathione peroxidase then sparks the conversion of reduced glutathione to oxidized glutathione, a free radical itself.

If there is sufficient glutathione in the cell, the newly unstable oxidized glutathione pairs with available glutathione with the help of an enzyme called glutathione reductase. This sends it back to reduced glutathione status and gets it ready to return to service protecting cells.

Glutathione recycling helps balance immune function and shield thyroid tissue from inflammation and autoimmune attacks. Glutathione also helps repair damaged tissues, such as in the case of leaky gut. Supplementing to increase glutathione levels – the RedRiver clinical research observations Between all of our clinics, we see several hundred patients each day, which puts us at an unparalleled advantage when it comes to honing in on the best support for patients. Below is an overview of what’s available to boost glutathione levels and what we have seen work the best for our patients. Glutathione recycling Glutathione recycling is a good place to start to increase glutathione activity inside of cells. A variety of nutritional and botanical compounds have been shown to support glutathione recycling, such as the following: N-acetyl-cysteine, which quickly metabolizes into intracellular glutathione. L-glutamine, which helps generate glutathione. Alpha-lipoic acid, which recycles and extends the life span of vitamin C, glutathione and coenzyme Q10, all of which are needed for glutathione recycling. Selenium, which is a cofactor for the enzyme glutathione peroxidase that converts reduced glutathione to oxidized glutathione to protect cells. Milk thistle, which significantly increases glutathione and improves the ratios of reduced and oxidized glutathione. Gotu kola, which increases glutathione peroxidase and glutathione in general. Cordyceps, which supports glutathione synthesis and activates the glutathione enzyme cycle. Taken together, these botanicals and compounds activate the glutathione peroxidase and reductase enzymes to promote a healthy glutathione recycling system.I use a stand-alone product to support glutathione recycling containing all of these ingredients called Glutathione Recycler by Apex Energetics. This product was developed by Dr. Datis Kharrazian, who pioneered our modern understanding of Hashimoto’s and glutathione.We have found this product works best when used in conjunction with a liposomal glutathione blend, which I discuss below. Liposomal glutathione Stock image | Photo by Shidlovski/iStock/Getty Images Plus, St. George News We are very impressed with patients’ results from a liquid liposomal glutathione blend called Trizomal Glutathione by Apex Energetics, also formulated by Kharrazian. Trizomal Glutathione provides both bioactive glutathione and the glutathione precursor N-acetyl-cysteine, meaning it’s for comprehensive glutathione support.Although dosages vary depending on the degree of inflammation, we generally start people […]


Memory-making involves extensive DNA breaking

Memory-making involves extensive DNA breaking

The urgency to remember a dangerous experience requires the brain to make a series of potentially dangerous moves: Neurons and other brain cells snap open their DNA in numerous locations — more than previously realized , according to a new study — to provide quick access to genetic instructions for the mechanisms of memory storage.

The extent of these DNA double-strand breaks (DSBs) in multiple key brain regions is surprising and concerning, says study senior author Li-Huei Tsai , Picower Professor of Neuroscience at MIT and director of The Picower Institute for Learning and Memory, because while the breaks are routinely repaired, that process may become more flawed and fragile with age. Tsai’s lab has shown that lingering DSBs are associated with neurodegeneration and cognitive decline and that repair mechanisms can falter .

“We wanted to understand exactly how widespread and extensive this natural activity is in the brain upon memory formation because that can give us insight into how genomic instability could undermine brain health down the road,” says Tsai, who is also a professor in the Department of Brain and Cognitive Sciences and a leader of MIT’s Aging Brain Initiative . “Clearly, memory formation is an urgent priority for healthy brain function, but these new results showing that several types of brain cells break their DNA in so many places to quickly express genes is still striking.”

Tracking breaks

In 2015, Tsai’s lab provided the first demonstration that neuronal activity caused DSBs and that they induced rapid gene expression. But those findings, mostly made in lab preparations of neurons, did not capture the full extent of the activity in the context of memory formation in a behaving animal, and did not investigate what happened in cells other than neurons.

In the new study published July 1 in PLOS ONE , lead author and former graduate student Ryan Stott and co-author and former research technician Oleg Kritsky sought to investigate the full landscape of DSB activity in learning and memory. To do so, they gave mice little electrical zaps to the feet when they entered a box, to condition a fear memory of that context. They then used several methods to assess DSBs and gene expression in the brains of the mice over the next half-hour, particularly among a variety of cell types in the prefrontal cortex and hippocampus, two regions essential for the formation and storage of conditioned fear memories. They also made measurements in the brains of mice that did not experience the foot shock to establish a baseline of activity for comparison.

The creation of a fear memory doubled the number of DSBs among neurons in the hippocampus and the prefrontal cortex, affecting more than 300 genes in each region. Among 206 affected genes common to both regions, the researchers then looked at what those genes do. Many were associated with the function of the connections neurons make with each other, called synapses. This makes sense because learning arises when neurons change their connections (a phenomenon called “synaptic plasticity”) and memories are formed when groups of neurons connect together into ensembles called engrams.

“Many genes essential for neuronal function and memory formation, and significantly more of them than expected based on previous observations in cultured neurons … are potentially hotspots of DSB formation,” the authors wrote in the study.

In another analysis, the researchers confirmed through measurements of RNA that the increase in DSBs indeed correlated closely with increased transcription and expression of affected genes, including ones affecting synapse function, as quickly as 10-30 minutes after the foot shock exposure.

“Overall, we find transcriptional changes are more strongly associated with [DSBs] in the brain than anticipated,” they wrote. “Previously we observed 20 gene-associated [DSB] loci following stimulation of cultured neurons, while in the hippocampus and prefrontal cortex we see more than 100-150 gene associated [DSB] loci that are transcriptionally induced.”

Snapping with stress

In the analysis of gene expression, the neuroscientists looked at not only neurons but also non-neuronal brain cells, or glia, and found that they also showed changes in expression of hundreds of genes after fear conditioning. Glia called astrocytes are known to be involved in fear learning, for instance, and they showed significant DSB and gene expression changes after fear conditioning.

Among the most important functions of genes associated with fear conditioning-related DSBs in glia was the response to hormones. The researchers therefore looked to see which hormones might be particularly involved and discovered that it was glutocortocoids, which are secreted in response to stress. Sure enough, the study data showed that in glia, many of the DSBs that occurred following fear conditioning occurred at genomic sites related to glutocortocoid receptors. Further tests revealed that directly stimulating those hormone receptors could trigger the same DSBs that fear conditioning did and that blocking the receptors could prevent transcription of key genes after fear conditioning.

Tsai says the finding that glia are so deeply involved in establishing memories from fear conditioning is an important surprise of the new study.

“The ability of glia to mount a robust transcriptional response to glutocorticoids suggest that glia may have a much larger role to play in the response to stress and its impact on the brain during learning than previously appreciated,” she and her co-authors wrote.

Damage and danger?

More research will have to be done to prove that the DSBs required for forming and storing fear memories are a threat to later brain health, but the new study only adds to evidence that it may be the case, the authors say.

“Overall we have identified sites of DSBs at genes important for neuronal and glial functions, suggesting that impaired DNA repair of these recurrent DNA breaks which are generated as part of brain activity could result in genomic instability that contribute to aging and disease in the brain,” they wrote.

The National Institutes of Health, The Glenn Foundation for Medical Research, and the JPB Foundation provided funding for the research.


Nutrition deficiencies can impact your mental health

Nutrition deficiencies can impact your mental health

Mental and physical health can be boosted with a nutrient-dense, healthy diet. Many times, low levels of essential nutrients can cause a feeling of low and can be linked to depression, irritability, and anxiety. “One must understand the signs of nutrition deficiencies. Mood disorders can be caused by various factors such as psychological, biological, genetic, environmental, and circumstantial. Nutritional imbalance happens to be the most neglected biological factor for mood swings/disorders. Very few people emphasise the connection between nutrition and depression, while most of them easily understand the connection between nutritional deficiencies and physical health. One’s vitamin, mineral and key nutrition deficiencies can compromise optimal brain functioning and increase irritability, tiredness, and depression,” says Eshanka Wahi, Culinary Nutrition Coach.
While deficiencies are extremely unique to each individual, below are 7 nutrition inclusions that Eshanka recommends to boost mental and physical health. If your diet doesn’t comprise of nutrient-rich foods she recommends to take additional supplements for those listed below:

1. Vitamin D
Vitamin D regulates the production of adrenaline, noradrenaline, and dopamine, and plays a vital role in hormonal balance. Vitamin D deficiency is linked with the presence of an active mood disorder. Mild Alzheimer’s, altered sleep patterns and mood issues, and fatigue are common disorders. To make one’s diet rich in vitamin D natural sources such as eggs, fatty fish, and mushrooms and fortified foods like milk, flour, rice, cheese, and oats should be consumed.
2. Vitamin B (B1, B6, B7, B12, B complex)
People with B vitamin deficiencies experience depression, anxiety, and mood swings. Folate is at the forefront of mood management. People fighting depression have lower levels of folate in the blood. Folate is present in green leafy vegetables, beans, peas, peanuts, and other legumes, and citrus fruits.

3. Magnesium
Magnesium levels have a negative correlation with the occurrence of depression. The deficiency of magnesium is known to increase the occurrence of many mental syndromes like agitation, anxiety, irritability, confusion, asthenia, sleeplessness, headache, delirium, hallucinations, and hyperexcitability. Therefore, one must include magnesium-rich foods such as pumpkin seeds, almonds, peanuts in their everyday diet

4. Iron
Iron Aids in the making of red blood cells, that carry oxygen around the body, and iron deficiency can lead to poor concentration, decrease in cognition (attention span, intelligence, and sensory perception functions), anxiety, irritability, and depression. The prolonging lack of iron can lead to headaches and breathlessness too. That is why choosing the right combination of food choices is the key. For instance, if one consumes a palak paneer and thinks that it will give enough iron, then they are mistaken. The calcium present in paneer limits the absorption of iron in spinach when consumed in combination. Instead, pair iron-rich sources with vitamin C. For example, food pairing to increase nutrient absorption includes Spinach with lemon juice. Iron capsule to be consumed with lemon water to increase nutrient absorption.

5. Selenium
The lower the level of selenium in the diet the more reports of anxiety, depression, and tiredness. Higher selenium levels are directly associated with lower depressive symptoms. The best food source of selenium is Brazil nuts

6. Omega-3 Fatty Acids
Omega-3 fatty acids are vital for brain function, especially memory and mood. If your diet is low in good quality fats, like omega-3s, then your body can only make low-quality nerve cell membranes. Oily fish like salmon and tuna are great sources of omega-3 fatty acids, as are fish like cod and cod liver oil. These healthy fats can also be found in flaxseeds and walnuts. Extra Omega 3 Fish Oil supplements are recommended.

7. Zinc
Zinc, another essential mineral that regulates the brain and body’s response to stress. It is the brain where zinc is found in the highest concentration in our body, making it central to healthy brain function. It isn’t just responsible for activating your central and peripheral nervous system but is also required for neurotransmitter, enzymatic and hormonal processes. Zinc deficiency does result in anxiety, schizophrenia, and eating disorders. Rich sources of zinc include meat, poultry, oysters, spinach, pumpkin seeds, raisins, and dark chocolate.